Uas ua tsis tiav txoj cai nras ceg thaiv

Nyob rau hauv no tsab xov xwm peb xav txog qhov no pathology li tsis tiav blockade ntawm txoj cai ceg nras ceg block. Qhov no ib nrab ua txhaum ntawm hluav taws xob impulses nyob rau hauv lub plawv. Tus kab mob no yog feem ntau asymptomatic, tab sis pov thawj ntawm cov organic kab mob plawv thiab feem xyuam rau txoj lub sij hawm ntawm cov thawj kab mob. Tus kab mob yog raws li nyob rau hauv cov kev tshwm sim ntawm electrocardiographic kev tshawb fawb. Uas ua tsis tiav txoj cai nras ceg thaiv yog ib tug thiab ib tug ib nrab rau ob tag nrho plaub-kaum feem pua ntawm cov neeg mob, thiab nrog lub hnub nyoog, cov nuj nqis tsuas nce. Ntawm tag nrho cov hom ntawm blockades nyob rau hauv zaus no pathology yog ranked thib ob.

Etiology ntawm nws tshwm sim

Yog vim li cas muaj peev xwm pab tau raws li lub plawv tsis xws luag, coronary artery kab mob thiab ib tug overdose ntawm anti-arrhythmia, pulmonary lub plawv, stenosis ntawm lub mitral valve, pulmonary tawg, tricuspid plawv, mob heart attack myocardial infarction, myocarditis, cardio, hypertensive plawv, npaus ntawm cov electrolytes nyob rau hauv lub cev, thiab txoj cai ventricular hypertrophy. Heev tsawg ua tsis tiav txoj cai nras ceg thaiv kuaj nyob rau hauv kev noj qab nyob cov tub ntxhais hluas. Nyob rau hauv xws li mob, peb yuav tau tham txog yog li qub.

Uas ua tsis tiav txoj cai nras ceg thaiv yog qhov ua rau deceleration kis ob qhov ntswg thiab ectopic pulses txij ntawd lawm. Qhov no, nyob rau hauv lem, ua rau yus mus rau ib tug drastic hloov nyob rau hauv lub excitation sequences ntawm ib co ntawm qhov chaw ntawm lub ventricles los yog cia li kiag li ventricle.

Uas ua tsis tiav blockade cai nras ceg thaiv: cov tsos mob

Nyob rau hauv Feem ntau, qhov no pathology tsis ua kom muaj hnov cov tsos mob, vim hais tias cov atherosclerosis thiab zaus ntawm lub plawv tsis cuam tshuam.

Mob ntawm tus kab mob no, raws li hais saum toj no, yog raws li nyob rau hauv cov ntaub ntawv tau los ntawm txoj kev electrocardiographic kev tshawb fawb.

Ntawm no yog ib daim ntawv teev cov cim qhia tias yog pom nyob rau nws:

• QRS complex ntev yog nce me ntsis, mus txog cuaj los kaum ib hundredths ntawm ib tug thib ob.
• Nyob rau hauv thawj thiab V6 ua me ntsis widened hniav S thiab txoj cai mis V1 abduction QRS complex nruab nrab rSr, los yog rsR.

Uas ua tsis tiav blockade cai nras ceg thaiv: kev kho mob

Lub ntev thiab stability ntawm cov uas twb pathology feem ntau tsis yuav tsum tau tej kev kho mob. Tab sis nyob rau hauv tej qhov chaw nws cov tsos coincides nrog lub aggravation kab mob plawv, raws sij hawm kev kho mob yog ib yam uas tseem ceeb heev rau lub normalization ntawm mob conduction. Yog hais tias ob qho tag nrho ob txhais ceg blockade lwm, ib ntus lus sib laws liag qhia, muab ua qhov ntev ntawm HV-luv tshaj 80-100 milliseconds. Tag nrho cov laus cov neeg mob uas conduction txawv txav xws li yog kev kawm mus rau yuav tsum pw tsev kho mob. Nyob rau hauv mob mob thiab nyob rau hauv lub qhaj ntawv ntawm contraindications tshuaj kho yuav tau mus thov, tsis nco qab lub inconstancy thiab unreliability ntawm xws txhais tau tias feem xyuam rau txoj no pathology. Nyob rau hauv qhov zoo tshaj plaws cov ntaub ntawv, koj yuav suav tau rau ib ntus nyhuv.

Lawv yog siv rau cov kev kho mob ntawm tej khoom:

1. Anticholinergics, uas txo vagal influences, e.g. 0.1% atropine ib milliliter intravenously thiab 0.2% platifillin raws li ib tug milliliter los yog raws li subcutaneously, intravenously, tab sis nyob rau hauv cov ntaub ntawv no nws yog tsim nyog los ua kom paub 5% zib nyob rau hauv volume los 500 ml.
2. Cov tshuaj uas ua rau kom cov yam ntxwv ntawm lub sympathetic mob conduction system, piv txwv li, 5% ntawm ephedrine nyob rau hauv ib tug milliliter intramuscular, subcutaneous, los yog tso dej raws leeg txhaj tshuaj, 0.2% norepinephrine, nyob rau hauv tus nqi ntawm 1 ml muab rau piam thaj, los yog 0.05% alupent, ib nrab mus rau ib lub voos xwmfab, muab ob intramuscularly thiab intravenously, los yog 0.1% izadrin nyob rau hauv ib tug milliliter volume.
3. Raws li siv glucocorticosteroid cov tshuaj hormones, e.g., hydrocortisone, ntawm tus nqi ntawm mus txog 200 mg ib hnub twg.
4. Yuav kom txo tau cov ntsiab lus ntawm poov tshuaj yog ntshaw kom siv 1% ntawm Lasix nyob rau hauv ib tug milliliter ntau.

Raug ntawm tus kab mob no yog heev paaj, tab sis, muab txoj kev pheej hmoo ntawm thab plaub rau lawv tag nrho cov xaiv ntawm cov yam mob cov kab mob, yuav tsum yog heev tiag rau nws kho mob.