Cerebral infarction

Cerebral infarction (ischemic stroke) muaj nyob rau hauv cov ntaub ntawv ntawm tseem ceeb yuav txo tau ntawm cerebral ntshav txaus. Ntawm cov kab mob uas ua rau nws txoj kev loj hlob, hauv thawj qhov chaw yog atherosclerosis, txav lub zoo cov hlab ntsha. Feem ntau sau tseg rau nws ua ke nrog arterial tawg, mob ntshav qab zib mellitus. Nyob rau hauv tsawg tus neeg mob, txawv arteritis, rheumatic fever, congenital plawv tsis xws luag decompensated, ntshav kab mob, kis kab mob, intoxication, provoked infarction. Cov ua ntawm tus kab mob zais nyob rau hauv lub hlwb thiab lub cev kev nyuaj siab. Feem ntau cov feem ntau raug tus neeg ntawm tsib caug rau caum xyoo los yog laus dua.

Tsim focal necrosis yuav tsum tau qhia nyob rau hauv sib txawv thaj tsam ntawm lub hlwb. Feem ntau lawv tshwm sim nyob rau hauv lub phiab ntawm nruab nrab leeg. cerebral infarction tej zaum yuav gray (dawb) los yog mixed liab. Feem ntau yog dawb. Provoking cerebral infarction pathogenetic mechanisms yog heev ntau haiv neeg. Cov no muaj xws vascular thrombosis, embolism, spasm. Kwv yees li ib nrab ntawm txhua tus neeg mob cerebral infarction nyob rau hauv lub keeb kwm yav dhau ntawm atherosclerosis sawv nyob rau hauv lub pathology ntawm lub extracranial vertebral thiab carotid cov hlab ntsha.

Ua ntej txoj kev loj hlob ntawm tus kab mob nraum lub plawv mob. Lawv feem ntau hu ua ischemic tawm tsam. Lawv tsiag ntawv los ntawm kev qhia txog lub tsis ruaj tsis khov focal cov tsos mob (paresthesia, hais lus ntshawv siab, paresis). Raws li ib tug txoj cai, lawv yog cov luv luv-term tsis muaj ntshav mov rau thaj tsam ntawm lub paj hlwb, uas tom qab npaj lub plawv nres. Nyob rau hauv tej rooj plaub, lub transistor ischemic tawm tsam feem ntau cia li ua ntej nws tsos. Yog li, lawv tsim lub tsev kho mob "prodrome" stroke. Tej zaum kuj muaj yog ib tug ntse mob nyob rau hauv lub taub hau, kev tsis xis nyob.

Ua ib tug ischemic stroke tau ntawm txhua lub sij hawm. nws feem ntau tshwm sim sai li sai tau tom qab licas los yog thaum mus pw. Nyob rau hauv ib co neeg mob, pib cov tsos mob yog txuam nrog rau lub xeev ntawm siab load, tus ntawm lub siab ntsws zoo tshaj, haus dej cawv, tej general, xws li kab mob kab mob los yog ntshav poob. Ischemic stroke yog feem ntau raws li los ntawm myocardial infarction.

Rau lub xeev ntawm cov feem ntau cov yam ntxwv yog cov nce ntawm ib ncig neurological tej yam tshwm sim gradual, tshwm sim li ob peb teev, thiab tej zaum ob los yog peb hnub. Nyob rau tib lub sij hawm tej zaum yuav muaj ib tug sawv ces undulating daim ntawv. Tej zaum cov neeg kawm ntawv uas heev ntawm lub nce, lub weakening. Nyob rau hauv tej rooj plaub, muaj apoplectiform lub xeev ntawm kev loj hlob.

Cerebral infarction. Kev Kho Mob.

Nws kim heev txhab yog yus muaj los ntawm ib tug heev heev mob ntawm cov neeg mob. Manifestation ntawm cov tsos mob focal yog nyob ntawm seb cov ntsha.

Therapeutic ntsuas thaum lub sij hawm infarct lub xeev ntawm lub paj hlwb yog nqa tawm nyob rau hauv daim ntawv ntawm intensive kev saib xyuas.

Plawv thromboembolism koom nrog koom haum saib xyuas ntawm heparin los yog tsawg molecular yuag derivatives (klivarina, fraxiparin). Tom ntej daim ntawv thov yog ua nyob rau hauv cov kev tswj ntawm cov ntshav txhaws.

Siv thiab antioxidants (tshuaj "Unitol" IM los yog e-vitamin), daim nyias nyias npaj ( "Essentiale", iv). Niaj hnub no lub cev qhuav dej intensive kev kho siv saluretikov, osmouretikov yuav luag yeej tsis siv vim hais tias ntawm inefficiency. Lub usefulness ntawm glucocorticoids yog ib co questionable.

Cov feem ntau metabolic los ntawm cov tshuaj siv. Cov no muaj xws tshuaj "Instenon" thiab "Aktovegin". Nyob rau hauv no aerobic ua pa yog tshuab txais nyob rau tom qab ntawm hypoxia thiab ischemia los ntawm pab oxygen siv thiab piam thaj noj lipid peroxidation thiab lwm yam.