Noj qab haus huv, Cov kab mob thiab mob
Hyperuricemia - yog dab tsi? Hom thiab kev kho mob
Ua lub theem ntawm uric acid nyob rau hauv cov ntshav ntawm tus txiv neej classed raws li hyperuricemia. Yuav ua li cas yog nws? Qhov no yog qhov tshwm sim ntawm purine metabolism mob, feem ntau tshwm sim los ntawm ib puag ncig yam tseem ceeb (khoom noj khoom haus thiab lwm tus), thiab caj yam. Qhov no pathology tau attracted xim tom qab rov ntsuam xyuas, uas qhia nws cov cawv nyob rau hauv lub chav kawm ntawm cov hlab plawv tus kab mob. Nws kuj xam tau tias yog ib tug leading biochemical tej yam tshwm sim ntawm gout. Hyperuricemia yog feem ntau asymptomatic, yog li ntawd yog tsis ib txwm kuaj tam sim ntawd.
Thaum nws yog hyperuricemia?
Nyob rau hauv thaum ntxov ua sawv ntawm mob ntawm purine metabolism muaj mob raum, ua ntej ntawm kev tawm tsam ntawm gouty mob caj dab thiab lwm yam kev mob. Qhov tseeb hais tias ob lub raum yog thawj muaj nyob rau hauv cov nyiaj them txheej txheem ib qho dhau heev lawm ntawm uric acid synthesis, nce lub qub feem ntawm urate, thiab hais tias muaj txhawb rau txoj kev pheej hmoo ntawm crystallization ntawm cov ntsev nyob rau hauv lub ob lub raum. Nce feem (qee) ntawm uric acid muaj ib tug ua ntxim rau cov tubules, interstitium ntawm raum, txhawb txoj kev loj hlob ntawm cov kab mob xws li hyperuricemia thiab hyperuricosuria. Tus thawj pathological mob tshwm sim los ntawm ntau zog concentration ntawm uric acid nyob rau hauv cov zis, uas yog tshwm sim los ntawm ib tug ua txhaum ntawm purine metabolism vim kev noj cov zaub mov nplua nuj nyob rau hauv purine bases, high-protein noj cov zaub mov, haus dej haus cawv kev tsim txom. Qhov thib ob qhia cov biochemical tsom xam ntawm cov ntshav.
hom hyperuricemia
Hyperuricemia yog thawj thiab. Tus thawj feem ntau yog tshwm sim los ntawm thawj gout, tsev neeg thiab cov kev tshuaj ntsuam genetic abnormality ntawm purine metabolism (konstitutsialny dispurinizm). Raws li cov causal yam yog muab faib ua peb yam:
- metabolic hom, vim qhov kev nce nyob rau hauv lub endogenous synthesis ntawm purines thiab nyob rau kev kub ceev urikozurii thiab purification kev lom ntaub so ntswg thiab kua nruab nrog cev (clearance) uric acid;
- raum hom, mob tshwm sim los ntawm lub raum feem ntawm uric acid thiab yus tsis muaj clearance;
- mixed yam, uas yog ib tug ua ke ntawm ob tug thawj tej yam kev mob nyob rau hauv uas uraturia txo los yog tsis ntev, thiab hauv av clearance yog unchanged.
cov tsos mob
Nyob rau hauv xyoo tsis ntev los, feem ntau nyob rau hauv lub chav kawm ntawm ib tug kev xeem thaum lub sij hawm ntawm cov ntshav nyob rau hauv biochemical tsom xam mob hyperuricemia. "Yuav ua li cas yog nws?" - tus thawj lo lus nug nug los ntawm cov neeg mob, vim muaj cov tsis muaj tej yam tshwm sim ntawm tus kab mob tsis pom. Tus kab mob yog, tseeb, feem ntau kis tau yuav luag asymptomatically.
Yuav ua li cas txawm xws unexpressed hyperuricemia, uas nws cov tsos mob thiab lawv ces nyob rau hauv feem ntau uas tsis yog-kev? Nyob rau hauv cov me nyuam, qhov no pathological mob yuav qhia los ntawm cem quav, mob plab, nocturnal enuresis, logoneurosis, lub suab qw, tawm fws. Cov me nyuam hluas feem ntau tshwm sim xws tsos mob ntawm hyperuricemia li rog, mob nyob rau hauv lub lumbar cheeb tsam, khaus nyob rau hauv lub qhov zis, biliary dyskinesia. Los ntawm kev soj ntsuam daim duab yuav nrog intoxication thiab asthenia. Nyob rau hauv neeg laus, nws yog tsim nyob rau hauv ib thaum ntxov rau theem ntawm tus kab mob loj hlob interstitial nephritis. Nws yog tau mutate rau hauv pyelonephritis theem nrab hom los ntawm cov kev txiav txim ntawm ib tug kab mob tus kab mob, tsim dej siab tej yam kev mob rau cov tsim ntawm raum pob zeb. Nws yog tsis yooj yim dua, thiab lub raum pob zeb, los yog nephrolithiasis. Lub hauv paus ntawm lub tsim los ntawm cov urinary pob zeb yuav tsum nco ntsoov cov nram no metabolic mob: modifying lub acidity ntawm cov zis, hypercalciuria, hyperoxaluria, giperfosfaturiya, giperurikuriya thiab hyperuricemia. Hyperuricemia yog feem ntau yog feem ntau txuam nrog rau ntau yam pathologies ntawm lub digestive ib ntsuj av.
muaj tej yam
Kab Mob tsim nyob rau hauv keeb kwm yav dhau ntawm lub ceev tsim ntawm uric acid, feem ntau yog tshwm sim los ntawm tej yam tseem ceeb:
- kev koom tes nyob rau hauv lub purine metabolic dab;
- deterioration ntawm lub raum muaj nuj nqi;
- siab cov ntsiab lus ntawm fructose nyob rau hauv cov khoom noj.
Ua rau hyperuricemia
Lub ntsiab ua rau tus mob no yog siv tsis raws cai ntawm khoom noj khoom haus, nyeem purines, zaub mov muaj roj. Tsis muaj tsawg txaus ntshai starvation thiab kev puas tsuaj ntawm ntaub so ntswg, qog phem. Tej zaum pab mus rau txoj kev loj hlob ntawm hyperuricemia cov kab mob ntawm lub lymphatic system, ntshav.
kev kho mob
Lub deterioration ntawm lub pom zog thiab mob ntawm lub raum tubular zog yog tus txhais provoking pathology xws li hyperuricemia. Yuav ua li cas yog nws rau lub xeev, nws yog ib qho pub los yog kis tau? Nrhiav mob yog xav paub ntau ntau nyob rau hauv cov neeg laus raws li ib tug tshwm sim ntawm sclerosis ntawm lub raum hlab ntsha. Hyperuricemia yog feem ntau kuj satellite pathologies xws li anemia, mob eczema, acidosis, psoriasis, toxemia nyob rau hauv cev xeeb tub.
Installation mob "hyperuricemia" kev kho mob yog muab raws li tus tau txais cov ntaub ntawv kuaj kev tshawb fawb thiab lwm yam ntxiv soj ntsuam. Nws cov hauv paus yog lub zaub mov kev kho mob. Los ntawm cov neeg mob txoj kev noj haus zaub mov cais cov khoom noj uas muaj ib tug tseem ceeb npaum li cas ntawm purine derivatives, los yog lawv siv yog tshuam txo. Medical Hoob no muaj cov urikozodepressornye tshuaj, tshuaj nrog uricosuric kev ua si. Ib qho tseem ceeb nam ntawm kev kho mob yog pom tias yuav tau qhov kev kawm ntawm alkaline zis. Self-kev kho mob yog tsis tsim nyog, txawm noj cov zaub mov tsim nyob rau hauv tus neeg lub tswv yim, nyob rau hauv thiaj li yuav tiv thaiv kom txhob muaj ib tug ntawm cov loj teeb meem - gout.
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