Noj qab haus huvCov kab mob thiab mob

Erysipelas kab mob. Soj ntsuam kev tshwm sim thiab kev kho mob

Erysipelas - ib tug kab mob ntawm cov kab etiology uas muaj feem xyuam rau ntawm daim tawv nqaij. Feem ntau tshwm sim ntawm cov poj niam nyob rau hauv 60% ntawm cov neeg tsim nyob rau hauv cov neeg tshaj 40 xyoo. Nyob rau hauv tas li ntawd, rau qhov kab mob no yog yus muaj los ntawm ib tug ntshiab seasonality - exacerbations tshwm sim nyob rau hauv lub caij ntuj sov thiab lub caij nplooj zeeg.

Erysipelas: etiology

Erysipelas kab mob no tshwm sim thaum noj kab mob (Streptococcus) exogenous los yog los ntawm cov kab mob muaj nyob rau hauv lub hloov lwm lub tsev los ntawm cov lymphatic thiab lub plawv systems. Nws yog ib nqi sau cia hais tias kev kho nyob rau hauv niaj hnub tshuaj thiab antiseptics yog ib theem siab, yog li no pathology yog tsis tshua muaj.

Txoj kev loj hlob ntawm tus kab mob ua rau kom lub scrapes thiab doog, tus xub ntiag ntawm venous ulcers, thrombophlebitis, varicose leeg, erysipelas feem ntau tshwm sim tom qab mob siab heev hnub raug los yog hypothermia nyob rau hauv cov neeg mob uas txhab los ntawm fungi, raws li zoo raws li lub xub ntiag ntawm kev txawv txav ntawm lub siab thiab lub plawv. Nws yuav tsum tau muab sau tseg tias erysipelas kab mob tshwm sim ntawm cov me nyuam nyob rau hauv cov neeg pluag nyob tej yam kev mob thiab koj tus kheej kev tu cev.

Causative ua hauj lawm ntawm tus kab mob no yog tus kab mob Streptococcus, txawm hais tias tom qab tshuab txais thiab staphylococcal muaj, uas yog kev koom tes nyob rau hauv lub pathological txheej txheem.

Rozsa: Cov tsos mob

Prodromal lub sij hawm ntawm los ntawm ob peb teev mus rau tsib hnub. Nyob rau hauv feem ntau tus neeg mob, tus kab mob no pib acutely.

Thawj zaug sij hawm ntawm manifested cov tsos mob ntawm intoxication. Tom qab ntawd tsis txaus siab muaj raws li nram no:

  • general tsis muaj zog thiab ua daus no;
  • mob taub hau thiab mob ib ce ;
  • nyob rau hauv 30% ntawm cov neeg xeev siab thiab ntuav;
  • yus ib tug kub (40 ° C);
  • nyob rau hauv lub cuam tshuam qhov chaw cim bursting, hlawv mob ntawm tsis muaj kev siv.

Nyob rau hauv lub neej yav tom ntej, tus kab mob manifests mug yam ntxwv daim tawv nqaij qhov chaw mob. Muaj yog ib tug me me liab chaw, uas muaj twb tau transformed rau hauv ib yam ntawm cov erysipelas, nrog fuzzy npoo nyob rau hauv ob peb teev. Cov tawv nqaij uas yog xim liab, o, moderately mob rau palpation. Kom effusion tsam inflammatory kev hloov ua rau lub tsim ntawm bubbles uas yog tsim nyob rau tom qab qhib ntawm lub qhov txhab. Yog hais tias cov npuas nyob twj ywm lawm, ces tom qab ntawd lawv qhuav li, txoj kev ua ib xim av ua kiav txhab.

Nws yuav tsum tau muab sau tseg tias lub ntsej muag yog ib tug kab mob yus muaj los ntawm kav ntev hloov nyob rau hauv daim tawv nqaij. Yog li ntawd, nyob rau hauv ib ob peb lub hlis tshuav pigmentation, thiab o ntawm daim tawv nqaij, raws li zoo raws li tuab thiab qhuav crusts, uas yog muab tso rau hauv qhov chaw ntawm bubbles.

Rozsa: kev kho mob

Kev kho mob ntawm txhab yog ua nrog cov lub ntiajteb txawj nqus txaus. Indications rau tsev kho mob yog: mob loj heev klinichekaya daim duab, nquag rov, muaj nyob rau hauv cov neeg mob uas muaj mob loj comorbidities, cov me nyuam thiab cov neeg laus.

Ib qho tseem ceeb qhov chaw nyob rau hauv cov kev kho mob ntawm erysipelas yuav siv sij hawm tshuaj tua kab mob. Nyob rau hauv tas li ntawd, muab anti-inflammatory siv tshuaj, vitamins. Cov neeg mob underwent detoxification thiab symptomatic txoj kev kho. Nws xav noj plawv tshuaj, diuretics, anti-kub cev tshuaj, prednisolone.

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