Nraum ischemic nres

Nraum ischemic nres, feem ntau yog tsim dheev hais txog tej focal tiv nujnqis regressed kiag li nyob rau hauv tsawg tshaj nees nkaum plaub teev. Qhov no txhais txawv breadth thiab ntau yam ntawm cov tsos mob. Nws yuav tsum tau muab sau tseg hais tias lub xeev tsis tau yeej ib txwm tau triggered thaum uas ischemia. Piv txwv li, migraine los yog epileptic syndrome nrog focal neurological ces. Txuag lub ischaemia cov tsos mob tshaj li ib teev twg yuav qhia tau tias ib tug cheeb tsam ntawm lub paj hlwb cov ntaub so ntswg infarction undergone.

Cov ces lub xeev ntawd hais tias tus txheej txheem muab kev koom tes nyob rau hauv lub pathological seem uas yog nkag nrog cov ntshav ntawm ib tug tej leeg (nruab nrab cerebral, carotid, thiab lwm yam).

Nraum ischemic nres muaj pathophysiological mechanisms. Nyob rau qhov no qhov tseeb hais tias tus stereotypical xwm, ntev thiab zaus ntawm recurrent lub sijhawm uas nws.

Piv txwv li, luv luv-lub sij hawm (kaum tsib los yog tsawg dua feeb), ua (tsib mus rau kaum lub sij hawm ib hnub twg), ib hom ntawm tsis muaj zog qaug dab peg nyob rau hauv tag nrho cov txhais tes los yog dab teg nrog lub xub ntiag (los yog tsis tuaj kawm ntawv) ntawm kev hais lus ntshawv siab tej zaum yuav qhia ib tug proximal narrowing los yog occlusion ntawm cov hlab ntsha, nrog tsis muaj peev xwm nyob rau hauv collateral ntshav mov. Qhov no mob ua rau yus focal ischemia nyob rau hauv lub cortex ntawm qhov opposite hemisphere. Txawm li cas los, nyob rau hauv lwm cov tes, ib tug rov ntawm kev hais lus tsis meej thiab tsis muaj zog nyob rau hauv qhov txhais tes los dab teg thaum lub sij hawm kaum ob teev yog cai nyob tsis tshua muaj ntshav txaus vim embolism. Ua ke nrog rau qhov no tej zaum txoj kev infarct cheeb tsam nyob rau hauv rau sab laug frontal lobe.

Mob ischemic stroke nyob rau hauv lub pas dej ua ke yog ib qhov tob tob cov hlab ntsha ua rau txoj kev loj hlob ntawm lub lesion, tsis tshaj ib tug centimeter. Lub xeev no yog denoted li cov transiently-lacunar ischemic nres. Taw ntsees nyob rau hauv lub basilar-vertebral system, nyob rau hauv rooj plaub yog hais tias lawv lub txiaj ntsim ntawm lub distal (ob sab sib dho) los yog proximal basilar leeg stenosis nyob rau hauv vertebrates, feem ntau manifested kiv taub hau no, dysarthria thiab ob lub zeem muag.

Yog li, nraum ischemic nres provoked los ntawm ob yam tseem ceeb: embolism thiab focal tee ntshav uas mov. Nyob rau hauv thawj cov ntaub ntawv, hauv lub xeev ntawm lub mechanism yog cuab kev. Yuav kom ib tus twg lesser kawm tshwm sim tia tom qab lub zos yuav txo tau ntawm cov ntshav mov mus rau lub hlwb. Nyob rau hauv tag nrho cov ntau yam, kom txhob muaj inflow vim arterial stenosis los yog occlusion ntawm ib tug tseem ceeb degree. Ntawm kuj zoo kawg tseem ceeb yog cov tsis muaj collateral ntshav txaus rau lub cheeb tsam ntawm ischemia. Nyob rau hauv tas li ntawd, nws yog tsim nyog los noj mus rau hauv tus account lub elasticity ntawm cov hlab phab ntsa, cov ntshav viscosity thiab lwm yam. Nraum ischemic nres nyob rau hauv xws ntaub ntawv yog ntshai raws li muaj tseeb, uas yog, raws li lub sijhawm uas nws yog tsis embolic keeb kwm.

State regress kiag li. Txawm li cas los, lawv cov zoo li no ib tug ceeb toom txog qhov yuav mob stroke nyob rau hauv lub neej yav tom ntej. Yog li, lub pathophysiological mechanisms ntawm no ob lub xeev yuav tsum tau los ua ke. Nws yuav tsum tau muab sau tseg tias tus kws kho mob yog tsis muaj peev xwm muab kev pab rau cov neeg mob nrog ib tug nraum ischemic nres, tsis paub nws ua. Thaum tus mob no yog tsim nyog qhov chaw kawm ib txog kev kuaj mob.

Yuav tsum pw tsev kho mob raug cov neeg mob laus tshaj plaub caug-tsib xyoos thiab muaj kev tsis txaus siab tshaj yav dhau los tsib lub xyoos (tsis tau noj mus rau hauv tus account lub hnub nyoog). Therapy yog qhia rau cov hlab plawv tus kab mob, nrog rau atherosclerosis, tom qab tsis muaj ntau tshaj li ob hnub ntawm lub caij uas muaj nraum ischemic nres. Kev kho mob yog nqa tawm nyob rau hauv lub tsev kho mob tom qab cov kev tshawb nrhiav kev ua ub no nyob rau hauv thiaj li yuav paub meej tias cov syndrome. Therapy aims tiv thaiv tsis tshua mob heev raws li txoj cai, raws li tau zoo raws li qhov tshwm sim ntawm tus mob stroke, plawv nres.