Cov tshuaj "Glidiab MV": cov lus qhia rau kev siv, tiag tiag

Rau tus txij nkawm txoj kev kho ntawm lub thib ob hom mob ntshav qab zib, nyob rau hauv uas insulin induces zus tau tej cov molecules raws li nyob rau hauv cov txhais tau tias siv gliclazide. Qhov no tshuaj yog ib tug sulfonylurea derivative teej tug mus rau lub thib ob tiam.

General yam ntxwv

Tshuaj "Glidiab MB 30" yog xav tau lub Lavxias teb sab sib npaug ntawm "Diabeton MV" Fabkis tshuaj. Qhov Tshuaj-Kws tsob nroj "Akrikhin" nyob rau hauv lub Moscow cheeb tsam.

Lub medicament koom qhov ncauj hypoglycemic cov neeg ua hauj nyob rau hauv cov ntsiav tshuaj daim ntawv muaj hloov tso tawm. Thas xim rau lawv cov qauv ib tug dawb los yog cream tej zaum yuav marble blotches. Ntsiav tshuaj zoo li tiaj thooj voos kheej kheej muaj ib tug chamfer.

Consumer pob yog ib tug pob. Nws tej zaum yuav muaj 30 los yog 60 ntsiav tshuaj fej nyob hlwv daim hlau.

qauv

Tsis zoo li cov tshuaj "Diabeton CF" nrog ib koob tshuaj 0,060 g gliclazide, y medicament "Glidiab CF" zoo tib yam nqi ntawm lub active ingredient nyob rau hauv lub ob zaug qis, uas yog 0,030 g

By tsaug zog tableting cov khoom xyaw muaj xws li hydroxypropylmethylcellulose, aerosilovye molecules, magnesium stearate, microcrystalline cellulose.

Muaj kuj yog ib tug yeeb tshuaj "Glidiab" nrog ib tug pa tso tawm ntawm lub active tshuaj. Ntau npaum ib ntsiav tshuaj 0.08 g ntawm gliclazide.

Yuav ua li cas puas

Txuas mus rau lub medicament "Glidiab MV" phau ntawv qhia no qhia txog qhov kev txiav txim ntawm gliclazide uas niamntiav stimulation ntawm insulin secretion nyob rau hauv β-hlwb, nyob rau hauv lub pancreas.

Nyob rau hauv tus ntawm cov ntsiav tshuaj insulinosekretornaya nce ua si ntawm lub qab zib molecules thiab peripheral ntaub so ntswg ua rhiab rau insulin lawm.

Nqaij glycogen, yog ib tug intracellular enzyme exhibits ntau dua efficiency. Muaj yog ib tug yuav txo tau ntawm lub luv los ntawm thaum pib ntawm txoj kev siv ntawm cov khoom noj kom tso lawm. Recovering insulin secretion nyob rau ntawm ib thaum ntxov ncov uas txawv los ntawm lwm yam sulfonylurea gliclazide precursors, uas yuav siv sij hawm qhov chaw nyob rau hauv lub thib ob theem. Txo postprandial qabzib.

Kev txhim kho microcirculation tshwm sim los ntawm combining platelet adhesion thiab cell normalizing vascular permeability, thiab txo txoj kev loj hlob ntawm atherosclerotic mikrotromboznyh dab, tus yuav txo tau kev tshua ntawm tej yam ntuj tso nyias mus nyias ntawm cov ntshav txhaws. Tsis muaj peev xwm teb cov lus receptor lug nyob rau hauv cov hlab ntsha nyob rau hauv lub adrenaline molecule.

Cov tshuaj yuav qeeb lub cim ntshav qab zib retinopathy theem, lub nonproliferative rau. Heev kev kho mob tus neeg saib xyuas raws li cov tej yam kev mob ntawm ntshav qab zib raum puas constituting qhov chaw lub luag hauj lwm rau filtering, nws yuav ua rau nws tau txo lub feem ntawm cov protein nyob rau hauv cov zis.

Cov tshuaj tsis nce tus luj ntawm lub cev, tab sis nyob rau hauv tsis tooj, decreases vim cov nyhuv rau hauv thaum ntxov theem ntawm insulin tso tawm. Nws tsis ntxias nce insulinemia.

Yog vim li cas thov

Cov kws kho mob pom zoo kom siv ntawm ib tug yeeb tshuaj nrog ib tug high school cov qab zib nyob rau hauv cov ntshav ntawm lub thib ob degree. Kev kho mob yog nqa tawm nrog me ntsis ntxim rau kev noj haus thiab hauj sim lub cev ua si.

Yuav kom medicament "Glidiab CF" indications txuam nrog kev tiv thaiv ntawm deterioration nyob rau hauv ntshav qab zib mob yus nephropathy, retinopathy, heart attack myocardial infarction thiab mob stroke.

Yuav ua li cas siv

Lub dosing txhais tau tias yog xaiv rau txhua tus neeg mob ib lub zuj zus, muab qhov tseeb ntawm tus kab mob, lub concentration ntawm qab zib ntawm ib qho kev npliag plab thiab tom qab 120 feeb tom qab noj mov noj.

Rau cov tshuaj "Glidiab MV" qhia ntawv nyob rau daim ntawv thov assigns thawj zaug txhua hnub koob tshuaj 0.03 g, uas yog sib npaug rau ib ntsiav tshuaj. Qhov no concentration yog muaj nyob rau hauv cov laus cov neeg mob tom qab lub hnub nyoog ntawm 65. Tshuaj siv lus ib ntsiav tshuaj nyob rau hauv thaum sawv ntxov thaum noj tshais.

Yog hais tias tsim nyog, lub ntau npaum si txhua txhua ob lub lis piam. Qhov siab tshaj plaws pub nyob rau hauv ib hnub mus siv li 0,120 g, sib nug xov mus rau 4 ntsiav tshuaj.

Lub medicament "Glidiab CF" siv nyob rau hauv qhov chaw ntawm lub tib lub npe nrog ib tug pa tso tawm cov tshuaj, siv 1-4 ntsiav tshuaj ib hnub twg.

Nws ua ke nrog txhais tau tias hypoglycemic txiav txim biguanide, alpha-glucosidase inhibitor ntawm lub insulin molecules.

Nyob rau hauv lub raum mob ua hauj lwm tsis muaj zog los yog muaj mob loj tsawv cim, thaum tus nqi ntawm creatinine clearance tsawg tshaj li 0.080 litres ib feeb thiab tsis txo cov dosing.

Thaum twg koj tsis tau

Ntsiav tshuaj "Glidiab MV" phau ntawv qhia tswv yim rau siv cov thawj hauv daim ntawv ntawm cov ntshav qab zib ntshav qab zib kab mob ntau dua ketones nyob rau hauv cov zis, paresis pais plab ntawm hyperosmolar ntshav qab zib coma thiab precoma rau loj tej kev phais thiab hlawv kev raug mob, kev puas dab thaum tsim nyog cov insulin kev kho mob.

Contraindications: mob loj heev mob ntawm daim siab los sis mob raum tau hauj lwm, plob tsis so tswj rhuav txhua, hloov cov haum ntawm zaub mov, qhov kev loj hlob ntawm ib tug hypoglycemic mob.

Koj muaj peev xwm siv tsis tau nyob rau hauv cov neeg mob uas kub taub hau, leukopenia, cev xeeb tub, mis-feeding thiab ntau intolerance ntawm cov khoom xyaw, cov tshuaj.

Ceev faj yog yuav tsum tau thaum xeem cov tshuaj, tshwj xeeb kev kho mob thiab xaiv ntawm noj tshuaj npaum cas rau cov neeg mob uas muaj haus dej haus cawv nyob tos thiab irregularities nyob rau hauv lub qog caj pas.

Nta ntawm kev kho mob

Los ntawm cov tshuaj "Glidiab MV" qhia rau cov ntsiab lus los yuav tsum tau mus muab nws nrog ib tug tsawg-calorie zaub mov noj cov zaub mov muaj ib tug uas tsis muaj carbohydrate cov ntsiab lus. Nws yuav tsum xyuas ntawm lub concentration ntawm qab zib nyob rau hauv cov hlab ntsha nyob rau hauv thaum sawv ntxov, ua ntej thiab tom qab ib tug noj mov.

Yog hais tias koj muaj mus phais los yog decompensation ntawm lub ntshav qab zib mob, tej zaum cov kev taw qhia ntawm insulin txhais tau tias.

Muaj lus ceeb toom gipoglikemichekogo txheej txheem tshwm sim nyob rau hauv lub siv ntawm ethyl cawv, ib tug uas tsis yog-tshuaj anti-inflammatory tus neeg saib xyuas thiab cov uas tsis muaj hwj chim. Haus dej haus cawv yuav ua rau disulfiramopodobnyh syndrome, nrog mob tej lub taub hau thiab lub plab mog, xeev siab thiab ntuav.

Nws yog tsim nyog yuav tau kho cov tshuaj ntawm tshuaj thaum lub sij hawm lub cev los yog kev nyuaj siab thiab untimely noj.
Tshwj xeeb tshaj yog rhiab rau cov teebmeem ntawm cov txhais tau tias yog cov neeg laus, cov neeg mob nrog kauj los yog txom nyem noj haus, debilitated cov neeg mob uas raug kev txom nyem los ntawm tsis muaj peev xwm ntawm adrenal-pituitary system.

Nyob rau hauv thawj zaug rau theem ntawm daim ntawv thov ntawm cov tshuaj nyob rau hauv lub xaiv ntawm ntau npaum, yog hais tias muaj yog ib tug predisposition mus hypoglycemic ntsoog, nws tsis yog tsim nyog los ua tej yam uas yuav tsum nce xim thiab ceev ceev psychomotor tshua.

phiv tshua

Los ntawm txoj kev "Glidiab MV" qhia muaj cov ntaub ntawv nyob rau hauv kev ua txhaum nyob rau hauv lub endocrine kabmob nyob rau hauv tsis ua hauj lwm nyob rau hauv cov ntsiav tshuaj hom, kev siv thiab kev txom nyem noj haus. Feem ntau, yuav txo tau cov zib nyob rau hauv cov hlab ntsha ua rau cov tshwm sim ntawm lub taub hau mob, qaug zog, tshaib plab, helplessness, ntxhov siab vim yog instantaneous tsis muaj zog txaus, aggression, tsis concentration, depressive lub xeev. Kuj muaj cov kev hloov ntawm nrig txog kev pom kev xaav, co, kov thiab convulsive tsis meej, kiv taub hau, gipersomniyai, ntiav ua tsis taus pa, tsis lub plawv dhia.

Digestive kabmob ua malfunctions nyob rau hauv daim ntawv ntawm cov malabsorption, xeev siab, raws plab, tsis muaj qab los noj mov anorexic, teeb meem ntawm ua tau hauj lwm mob hlwb, cholestatic daj ntseg, ua rau kom lub efficiency ntawm transaminase enzymes.

Undesirable dab nyob rau hauv lub hematopoietic system txuam nrog ib tug txo nyob rau hauv hemoglobin thiab leukocyte tooj platelet hlwb.

Cov tshuaj yuav ua kom ua xua nyob rau hauv daim ntawv ntawm cov khaus, urticaria, makulo-papular pob.

ntev li cov kev taw qhia

Txuas mus rau ib tug txhais tau tias "Glidiab MV" qhia ntawv nyob rau ceev faj tawm tsam ib tug overdose, uas yog qhia los ntawm ib tug txo nyob rau hauv ntshav qab zib concentration. Yog hais tias ib tug muaj zog tshaj tshuaj tej zaum yuav tsim hypoglycemic coma.

Yuav kom tshem tawm ib tug neeg yog muab me ntsis rau noj zoo absorbed hydrocarbons, piv txwv li, ib daim ntawm qab zib. Thaum tus neeg tsis nco qab lawm, nws nkag tau rau hauv lub hlab ntsha 40 feem pua dextrose los yog piam thaj tov thiab hno mus rau hauv cov nqaij ntawm glucagon nyob rau hauv ib tug npaum li ntawm 1 mg. Yog hais tias tus neeg mob yog tsaug zog, ces nws yog yuam kom noj zoo absorbed hydrocarbons kom tsis txhob rov hypoglycemic nres.

Ua ke nrog nrog rau cov tshuaj

Lub hypoglycemic ua si ntawm lub medicament "Glidiab CF 30 mg" yuav kho kom zoo los ntawm qhia thaum uas tig mus retarder angiotensin converting enzyme thiab Monoamine oxidase hom receptor blocker formations beta adrenozavisimyh thiab H2 gistaminozavisimyh raws li cimetidine, thiab flukonazolovyh mikonazolovyh noj tshuaj, uas tsis yog-tshuaj anti-inflammatory cov neeg ua hauj phenylbutazone, indomethacin, diclofenac.

Cov nyhuv uas muaj zog raug ntsiav tshuaj siv clofibrate thiab bezafibrate, ethionamide antitubercular cov tshuaj los ntawm cov pab pawg neeg ntawm salicylates, indirect anticoagulant sib txuas ntawm lub coumarin qauv, anabolic steroids, cyclophosphamide, chloramphenicol, sulfonamides nrog ib lub caij nyoog zoo.

Cov tshuaj zoo txo qab zib nyob rau hauv cov ntshav ntshav los ntawm kev siv blockers ntawm tubular feem, ethyl cawv, acarbose, ib tug biguanide, insulin.

Txo hypoglycemic nyhuv ntsiav tshuaj ua barbiturates, tshuaj raws li epinephrine, clonidine, terbutaline, ritodrine, salbutamol, txawm phenytoin, inhibitors ntawm carbonic anhydrase enzyme hom acetazolamide, thiazide diuretics, cov tshuaj hormones ntawm lub qog caj pas, lithium-muaj tshuaj, cov tshuaj no kev.

ethyl cawv molecule muaj feem xyuam rau cov emergence of gliclazide disulfiramopodobnyh txheej txheem.

Ntsiav tshuaj Active tivthaiv ua untimely depolarization thiab ventricular myocardial stunning nqaij thaum ua ke nrog mob glycosides.

Beta adrenoblokatornye txhais tau tias klonidinovye, reserpine, guanetidinovye tshuaj npog tus soj ntsuam hypoglycemia.

Views ntawm cov neeg mob

Nws yog tsis tsuas txuas mus rau lub medicament "Glidiab MV" phau ntawv qhia. Xyuas tham txog dab tsi cov neeg mob xav txog cov hauj lwm zoo ntawm cov tshuaj. Qhov cuab yeej no yuav pab tus neeg yuav txo tau cov concentration ntawm qab zib rau ib txwm theem, thiab nyob rau hauv ua ke nrog nrog kev noj haus hloov cov duab ntawm tus neeg mob lub neej.

Cov neeg hais tias ib tug zoo feature ntawm cov ntsiav tshuaj yog nws yooj yim siv nyob rau hauv thaum sawv ntxov. Raws li koj yuav tsis nco qab qhov yuav tsum tau rau kev kho mob thaum lub sij hawm ib hnub.

Nyob rau cov tshuaj "Glidiab MV" xyuas muaj peev xwm yuav hnov thiab ib tug tsis zoo ua cim txuam nrog rau ineffectiveness ntawm no lub cuab tam. Feem ntau qhov no tshwm sim thaum lub tsis ncaj ncees lawm dosing thaum ib tug me me npaum li cas ntawm tshuaj.