Noj qab haus huvTshuaj

Koj peritonitis mob plab txaus ntshai?

Peritonitis plab mog yog ib tug kab mob ntawm ib qho kev inflammatory xwm, uas yog ib ncig, raws li lub npe implies, ncaj qha mus rau hauv lub peritoneal kab noj hniav. Kws txawj tam sim no qhia ob peb yam tseem ceeb yuav ua tau qhov teeb meem no, nrog rau tus tuaj txog ntawm kab mob mus rau hauv cov menyaum peritoneal kab noj hniav. Tom qab, cov kab mob muab sai heev, nrog rau cov tshwm sim hais tias muaj yog ib tug inflammatory txheej txheem. Nyob rau hauv no tsab xov xwm uas peb yuav tham txog dab tsi peritonitis mob plab kab noj hniav. Tsis tas li ntawd xav txog nws variants thiab qhov yooj yim txoj kev kho mob.

kev faib

Rau hnub tim, kws tshawb fawb tau pom ib tug ntau yam ntawm cov kab mob ntawm lub plab kab noj hniav. Lawv kev kho mob, cov tsos mob thiab tom ntej kev ncaj ncees yog heev uas zoo sib xws. Raws li kev xyaum qhia tau hais tias, ib co neeg mob thiab tag ntawm nws tus kheej teem lawv tus kheej lub misdiagnosis ntawm tus kab mob noj txawv. Txawm li cas los, nyob rau hauv no tsab xov xwm peb yuav kov tsuas yog ib, uas nws muaj nws tus kheej tej nta. Yog li ntawd, muaj ntau ntau hom ntawm peritonitis:

  • Lub thiaj li hu ua txawj peritonitis mob plab yus txuam nyob rau hauv cov kua tsam, uas nyob rau hauv lem zoo nkaus li vim rau lub siab mob. Feem ntau tus kab mob no thiaj paub tias yog nyob rau hauv qeb ntawm cov neeg uas siv kev lim ntshav.
  • Secondary peritonitis mob plab kab noj hniav feem ntau muaj vim txhua yam kev puas tsuaj. Qhov no yuav ua tau ib tug ntau yam ntawm mob nyob rau hauv lub mob huam (e.g., rwj, mos txwv lub qhov txhab, cholecystitis, etc.).
  • Purulent peritonitis mob plab kab noj hniav yog tsim vim lub thiaj li hu ua plab hnyuv paresis, uas yog tam sim ntawd raws li los ntawm outpouring ntawm cov hlab ntsha nyob rau hauv cov kab noj hniav nws tus kheej.

Symptomatology ntawm tus kab mob:

  1. Tsis tu ncua mob plab.
  2. Lub xub ntiag ntawm cov ntshav nyob rau hauv cov quav.
  3. Kub taub hau.
  4. Ib tug tseem ceeb tsis qab los noj mov.
  5. Nce tawm hws.
  6. Ntuav.
  7. Tsam plab.

kev kho mob

Yog hais tias koj pom tag nrho cov saum toj no cov tsos mob, koj yuav tsum tsis muaj kev ncua nrhiav kev pab los ntawm ib tug kev tsim nyog. Txwv tsis pub, yog tias koj yuav, lawv hais tias, mus ncua cov mus xyuas, lub txim uas tsis muaj cov feem ntau dej siab. Yog li, txoj kev kho implies ib tug ua tiav kev tshem tawm ntawm qhov chaw ntawm kab mob thiab lub tom ntej tshem tawm ntawm inflammatory dab. Nyob rau hauv qhov tseeb, yog koj ntseeg hais tias cov kws txawj, lub caij thiab cov hauj lwm zoo ntawm kev kho mob nyob lom zem ntau nyob rau hauv thawj ua, uas provoked tus kab mob no. Yog xav tau tshem ntawm ib tug me me daim ntawv ntawm tus kab mob no, feem ntau yog ib tug tshuaj tua kab mob yog muab rau koj. Thaum advanced theem ntawm tus kab mob tshuaj tua kab mob yog tsis zoo li mus rau tiv nrog nws cov nuj nqi. Nyob rau hauv tej lub sijhawm feem ntau yuav tsum pab phais kev cuam tshuam. Ntawm cov hoob kawm, nyob rau hauv cov ntaub ntawv no, lub rehabilitation txheej txheem yuav coj ib tug me ntsis lawm, tej zaum xav tau thiab ua raws li cov kho tshiab ntawm lub plab kab noj hniav. Txog lub caij nyoog kawg no, nws daws xwb ib tug tshwj xeeb.

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